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CV-19 Autopsies Show Blood Clots: A ‘Bacterial, Not Viral Disease’

Doctors in Italy, Germany and India who autopsied patients found many COVID-19 cases are actually BACTERIAL rather than a virus. Many cases point to thrombosis as cause of death. Recommended treatments should include blood thinners such as simple aspirin.

In Italy doctors who broke with the World Health Organisation (WHO) protocols were able to effectively treat COVID-19 once they recognised many cases were nothing other than “Disseminated intravascular coagulation” (Thrombosis).

Having discovered this diagnosis, the Italian Ministry of Health immediately changed the coronavirus treatment protocols and began to administer to their CV-19 positive patients Aspirin 100mg and Apronax. These patients began to recover and as a result of this new method, the Ministry of Health released and sent home more than 14,000 patients in a single day.

Italian doctors report success in combating COVID-19 with “antibiotics, anti-inflammatories and anticoagulants”.  Indeed, nothing more than ASPIRIN, is shown as very effective, indicating that this disease had been poorly treated.

Italian doctors affirmed this treatment after autopsies on deceased patients showed blood clotting as cause of death. According to Italian pathologists, “The ventilators and the intensive care unit were never needed.”

Meanwhile, doctors in India affirm CV-19 is most likely a bacterial disease – it does not appear to be a virus – which explains why the WHO protocols for treating the pandemic as a virus have been ineffective.

Dr Jaideep Dogra, MD and in-charge of CGHS in Jaipur and Dr Luvdeep Dogra, DM-F Nephrology at Osmania University is reporting that in India they are having most success when treating the disease as a normal bacterial pneumonia infection.

The Tribune India’ (May 24, 2020) in ‘Co-existence of coronavirus with bacterial pathogen a major cause of fatalities’ tells us that:

“This ‘Chlamydia pneumonia’ is present in several heart patients, and around 10 per cent of the general population in the dormant stage. When COVID-19 attacks a person— breaking the immunity quotient—a process of blood coagulation initiates in pulmonary arteries leading to a cardiac arrest or Hypoxia in which the patient succumbs to the dual diseases.” [1]

‘The Tribune India’ continues:

“Researching ‘Chlamydia pneumonia’ for over two years on heart patients in Rajasthan, Dr Jaideep also published a paper in the International Journal of General Medicine.

“Eighty-five per cent of Covid-19 patients having normal Influenza-Like Illness (ILI) symptoms get treated, while 10-15 per cent of patients develops sudden breathlessness and land in an emergency. In such patients, C.penumoniae probably gets activated when COVID-19 breaks down the patient’s immunity. This bacterial pathogen has a tendency, it gets activated, causes arterial thrombo (coagulopathy)”, they said.”

Quoting a postmortem study on COVID-19 victims done by Italy, they found:

“Autopsy studies on 38 subjects from two hospitals in Italy who died of COVID-19 were systematically analysed. A relevant finding of the presence of platelet-fibrin-thrombosis in small pulmonary arteries, which fits into the clinical context of a ‘Coagulopathy’ was present in a majority of these patients (86.8{154653b9ea5f83bbbf00f55de12e21cba2da5b4b158a426ee0e27ae0c1b44117}, 33 out of 38). And, this was the turning point in the management of COVID 19”.

“Similarly, ‘Happy hypoxia’ of COVID-19 disease in which young patients succumb suddenly as their hypoxia goes unnoticed can be explained by the autopsy reports from young soldiers of Korean War and Vietnam War reporting an incidence of 45-77{154653b9ea5f83bbbf00f55de12e21cba2da5b4b158a426ee0e27ae0c1b44117} of Coronary atherosclerosis in the mean age group of 26 years. This indirectly co-relates with the known observation that chlamydia pneumonia is present in young populations too”, they hypothesized.

“However, why only 10-15{154653b9ea5f83bbbf00f55de12e21cba2da5b4b158a426ee0e27ae0c1b44117} of patients develop sudden breathlessness and land in the emergency room is still an unsolved puzzle. Moreover, while on positive pressure ventilation the condition of these patients paradoxically worsened. While those who got simple non-invasive oxygen delivery did show recovery. We may argue that the ventilated patients were inherently sicker but then is there something beyond that too?” they explained.

“Evidence has been accumulating on the role of Chlamydia pneumoniae-a species of Gram-negative obligate intracellular pathogen (the link between virus and bacteria) which causes a spectrum of lower and upper respiratory tract infection in humans and has been implicated in etiology/ pathogenesis of artery blockage”, the duo said.

A widely used anti-bacterial drug Azithromycin for COVID-19 patients is giving positive results. Doctors assume it is controlling the virus, but this may be a wrong notion, Dogras said, adding, Azithromycin has long been used for ‘Chlamydia pneumonia.’

Hence, the action of this anti-bacterial drug may lie in not curing a deadly virus (SARS CoV 2) but killing Chlamydia and preventing the mortality associated with the disease, they opined.

If Azithromycin is administered in early stages of Corona infection in the optimum doses, the mortality risks of this dreaded disease may be minimised“, they further stressed.

‘Annals of Internal Medicine’ (6 May 2020) in ‘Autopsy Findings and Venous Thromboembolism in Patients With COVID-19’ reported on autopsies performed at a single academic medical center, as mandated by the German federal state of Hamburg. They affirmed the prevalence of thrombosis in deceased COVID-19 patients. [2]

As has been the case worldwide, the mortality rate is highest among the elderly with multiple comorbidities. The report stated all patients autopsied had:

“preexisting chronic medical conditions, such as obesity, coronary heart disease, asthma or chronic obstructive pulmonary disease, peripheral artery disease, diabetes mellitus type 2, and neurodegenerative diseases.”

They concluded:

The high incidence of thromboembolic events suggests an important role of COVID-19–induced coagulopathy.”

The German study strongly recommended autopsy as a means of increasing understanding of the disease. They wrote:

“Despite modern diagnostic tests, autopsy is still of great importance and may be a key to understanding the biological characteristics of SARS–CoV-2 and the pathogenesis of the disease.”

[1] ‘The Tribune India’ (May 24, 2020) in ‘Co-existence of coronavirus with bacterial pathogen a major cause of fatalities’

[2]See: ‘Autopsy Findings and Venous Thromboembolism in Patients With COVID-19’

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